TRANSLATIONAL PHYSIOLOGY Combined proteomic and metabonomic studies in three genetic forms of the renal Fanconi syndrome

نویسندگان

  • Annalisa Vilasi
  • Pedro R. Cutillas
  • Anthony D. Maher
  • Severine F. M. Zirah
  • Giovambattista Capasso
  • Anthony W. G. Norden
  • Elaine Holmes
  • Jeremy K. Nicholson
  • Robert J. Unwin
چکیده

Annalisa Vilasi,* Pedro R. Cutillas,* Anthony D. Maher,* Severine F. M. Zirah, Giovambattista Capasso, Anthony W. G. Norden, Elaine Holmes, Jeremy K. Nicholson, and Robert J. Unwin Ludwig Institute for Cancer Research and Department of Biochemistry and Molecular Biology and Centre for Nephrology and Department of Physiology, University College London, London; Department of Biomolecular Medicine, Division of Surgery, Oncology, Reproductive Biology, and Anaesthetics, Faculty of Medicine, Imperial College London, London; Clinical Biochemistry, Addenbrooke’s Hospital, Cambridge, United Kingdom; Chair of Nephrology, Second University of Naples, Naples; and Center of Mass Spectrometry, Proteomics, and Bioinformatics, Institute of Food Science, Consiglio Nazionale Delle Ricerche, Avellino, Italy

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Combined proteomic and metabonomic studies in three genetic forms of the renal Fanconi syndrome.

The renal Fanconi syndrome is a defect of proximal tubular function causing aminoaciduria and low-molecular-weight proteinuria. Dent's disease and Lowe syndrome are defined X-linked forms of Fanconi syndrome; there is also an autosomal dominant idiopathic form (ADIF), phenotypically similar to Dent's disease though its gene defect is still unknown. To assess whether their respective gene produc...

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The urinary proteome in Fanconi syndrome implies specificity in the reabsorption of proteins by renal proximal tubule cells.

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TRANSLATIONAL PHYSIOLOGY The urinary proteome in Fanconi syndrome implies specificity in the reabsorption of proteins by renal proximal tubule cells

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Dent-Wrong disease, an X-linked recessive disorder of the proximal tubules, presents with hypercalciuria, nephrocalcinosis, nephrolithiasis, renal insufficiency, low-molecular-weight proteinuria, rickets and/or osteomalacia. Dent and Friedman initially characterized the disorder in 1964 following studies of two patients with rickets who presented with hypercalciuria, hyperphosphaturia, proteinu...

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تاریخ انتشار 2007